Paul Harch MD - Hyperbaric Oxygen Therapy: Scientific Foundati

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Topic: Hyperbaric Oxygen Therapy: Scientific Foundation & Applications to Neurological Disease by Paul Harch MD

Conference: Neurodegeneration: The Impact of Environmental Insult - Grand Rapids, MI - October 4 - 8, 2017

Summary: This lecture is an overview of hyperbaric therapy in neurodegenerative diseases with a review of the literature. The lecture will begin with a presentation of the scientific foundations of hyperbaric therapy (HBT). HBT will first be defined historically and then scientifically as a combination therapy of increased pressure and increased oxygen whose mechanism of action is epigenetic modulation of gene expression and suppression, particularly those genes controlling inflammation. The activity of this therapy will be shown to be most prevalent in acute, subacute, and chronic wound conditions. The literature on etiology of neurodegenerative disease will be reviewed to show that neurodegenerative diseases result from a combination of genetic and cumulative environmental factors that generate central nervous system wounds. Many of these diseases are characterized by the end-products of inflammation/wounding, namely scar (multiple sclerosis-MS, chronic traumatic encephalopathy-CTE) or protein aggregation (Alzheimers, Parkinson's Disease-PD). Lastly, the literature will be reviewed on HBT in inflammation and neurodegenerative and developmental diseases, including Parkinson's Disease, dementia (Alzheimer's and others), amyotrophic lateral sclerosis (ALS), CTE, MS, and autism spectrum disorder (ASD).

Learning Objectives:
Learning objective #1: To learn and understand the historical definition of HBOT, its inadequacies, and the newer scientific definition of hyperbaric therapy as a combination therapy with gene modulation effects.
Learning objective #2: To learn and understand HBOT as a treatment of acute, subacute, and chronic wounds in any location in the body.
Learning objective #3: To learn and understand that neurodegenerative diseases result from a combination of genetic factors and cumulative environmental factors that generate central nervous system wounds. Inflammation will be shown to be a dominant pathophysiology in the wounding.
Learning objective #4: To learn and understand the positive and negative literature on HBOT in a variety of neurodegenerative and neurodevelopment diseases and the effects of HBOT on inflammation.

Time: 1 hr. 22 mins.

File Type: Down-loadable Zip file including MP3 audio file and PDF slides.

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